By H. D. Schlumberger (auth.), Dr. Leonard Eleazar Glynn, Professor Dr. Horst Dieter Schlumberger (eds.)
With the creation of antibiotics acute inflammatory sickness has ceased to be the dominant challenge as a rule scientific perform and its position is now more and more occupied via power inflamma tory sickness of which the rheumatic ailments represent an important workforce. lwo points of those ailments have to be consid ered, their aetiology and their pathogenesis. In a few respects the latter is extra vital given that even if the aetiology is understood, as for instance the haemolytic streptococcus in rheumatic fever, the mechanism through which the infecting agent accomplishes the advance of the lesions that characterise the illness are nonetheless principally unknown. nonetheless extra so is that this the case in rheuma toid arthritis and different continual inflammations the place the aetio logical brokers are unknown. In an try to make clear the pathogenic mechanisms concerned, sev eral makes an attempt were made to urge similar lesions in ex perimental animals, partially to check underlying hypotheses, and in part to supply attempt occasions for the trial of latest therapeu tic brokers. In view of the deficiencies within the present drug deal with ment of persistent inflammatory ailment there are lots of who suppose that this can be principally as a result of inadequacy of the types avail capable for try out purposes.
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